Tuesday, 17th April 2018.
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Tuesday, 17th April 2018
great time. Your backorder already on way to you, there tomorrow, unless your
address is RD where it’ll be the usual extra day.
Friday, 13th April 2018
Shipping News !
has left the factory:
Backorders to go out first, of course!
“PBD” orders received to 5 pm Tuesday 17th will join priority backorders list
treated with utmost urgency.
received after Tuesday will likely not be actioned until following Monday,
PLEASE stock up from this
shipment early even if you don’t need them for quite a while since a
lot of it has already gone, pre-sold, with the next shipment over a month
away, we will likely run out again!
Go to old notices: Archive: click!
Scroll below for today’s
“lactate testing tidbit”
Lactate testing tidbits…
Update: Friday, 13th April 2018.
Monday, 19th February 2018
Games Indoor Rowing!
I will put a wee lactate-testing story here
soon, 20 years on from my last go at racing on “the machine”!
In the meantime check out my lactate profile
building up for NZ Masters Games scrolling down here… http://www.tanita-nz.co.nz/My%20TANITA%20testimonials%20071117.htm#KJG
As at 13/4 I am still to post an update of
season end results … soon I promise! …biz has been flat-out, not much time
to waffle on! ;-)
Friday, 8th December 2017
Again, apology for my sport bias but it is
the one I am most familiar with from my youth and the principles are applicable
to many sports!
: PART ONE
When I came out of Phys Ed School in ‘83 I
went back to rowing a couple years later to experiment on myself with what
I’d learnt. On arriving, University
“Easter Tourney” rowing was not what it is today, feeling it mickey-mouse,
so I didn’t bother with it for 4 years, proving costly when trying to get
“back on the oar” with any kind of representative ambitions. Was a so-called adult student going in,
coming back to career hunting and rowing at 26.
I became increasingly fascinated with the
concept of Power-to-Weight as a determinant of performance. Soundly frequently defeated by about half a
dozen of the countries top Light-weights (<72.5 kg), being only a “medium-weight”
myself with a best racing (and health) weight of about 82kg, I had to also
contend with about a dozen ahead of me in the Heavy-weight Single Scull
category. Did okay with placings in
Intermediate and Senior events.
Noticed improved condition through a Season
coincided with becoming very trim with reduced weight (from around an
off-season 88kg, back in the day when there was such as off-season!). Was performance improvement just a factor
of improved cardio-vascular function or, and, if both, was it about
carrying less weight? … which had the most impact?
Then there was the phenomenon of Philippa
Baker and Brenda Lawson “pocket rockets” who for years
annihilated any truly heavy weight women at the NZ Champs and won individual
world titles in Light Weight and Under 23 respectively, and then took on
the world together reigning for several years in the Double Sculls.
It was their performance that inspired me
to create a power-to-weight performance table that gave predictive on-water
single scull times … (requested data for only on “even” courses, meaning no
tail-wind or current) … which was taken from one’s bodyweight and 2000m Ergometer
score. I put a survey to NZ’s top
rowers who kindly allowed use of their data (which could not personally
identify them anyway) and using mine and other modest performers the spread
on the table from average to superior was completed. It was developed on
old Concept2 B Models. I suspect the algorithm has changed somewhat from
those days so that for such a performance predictive table to be used today
it would need to be re-done for today’s “E” model and superior technology
in faster carbon-fibre skiffs. We were only just
transitioning from wooden oars in my-day!
The impressive German immigrant Herman Krutzman residing in Cambridge, himself an accomplished
sport scientist, Olympic level coach, and builder of skiffs branded in his own
name, Krutzman, was an incredible source of additional
“real” education for this “newbie” in hydrodynamics revealing the massive
increase in resistance (additional
force required from what is a modest “biological engine”) for the increased
surface area of a skiff due to increased weight.
If a variety of people today sent me their
best Single race time (again, even conditions only), with their Erg 2K PB,
and their bodyweight for their on-water performance I could modernize this predictive table. I have used it for good motivational effect
when I was coaching about 20 years ago!
want the old table as it
is, I will see if I can dig it out, drop me an email, or if you have a collection
of old NZ Rowing mags it appeared there around the late ‘80’s (think it
Then there was the spectator confusing sensational
Cohen and Sullivan, multi-World and 2012 Olympic Double Scull Champions,
medium-weights and average height (just like me!) that resumed an interest
in this Power-2-Weight thesis. When
they stood on the dais against those they had defeated they appeared
“little guys” at around 6 foot! They
also paced their races in a style which was uncommon (subject for another
“lactate tidbit” article one day!). They would appear to sit modestly in
the field to finish over the last 500m with a blistering sprint that put
fans on the edge of seats, exhilarating, as they carved through the field.
This “Power-2-Weight” subject had its original
roots way back to a particular graph in the now classic undergrad “Textbook
of Work Physiology” by the legends Astrand and Rodahl (2nd Ed, 1977) that lends to this
(above) “story” to this day.
Traditional graphs of VO2max show that the
heavier you are the greater likelihood that you have the highest VO2max.
However, if you put a spread of people from those with light bodyweight and
relatively low VO2max against heavier people with higher VO2max you do not
necessarily have a predictor of performance in times, or across a finish
line, in cycling, running, or on water events!
However! The lower graph shows you that you do have
a predictor of performance if you correct for bodyweight by a function of
This means that instead of the all too common
standard expressions of simple litres per minute
VO2max or milliliters per minute per kilogram, use milliliters per minute per kilogram
it evens out (or accounts) for the effect of bodyweight on VO2max leaving you
an assessment of just who has the better VO2max irrespective bodyweight!
Why this third formulation is not used as
one standard in High Performance sport I have no idea, maybe someone can tell
me. (Maybe I’ve been away from ivory towers too long! ;-) It has only been around as a clearer predictor
than straight VO2max or milliliters per kilogram since the leading-light
sport scientists of the ‘60’s! Sure,
Lactate Threshold is a better “fitness” indicator, but VO2max assessment
still has the place for predicting the ultimate performance capacity and
international competitive potential (rank) of an individual.
practically is the value of all this long commentary for you? How do you
use this to improve performance?
the subject for Part Two!
Will try and get onto this before or over
the Xmas break. If you don’t hear from
me before, or I you, YOU HAVE A GREAT XMAS!
KJ & ER Goodhew
BM&S Imports – lactate.co.nz
Thursday, 28th September 2017
Please excuse this writer’s bias …an old rower!
NZ Rowing has hard-worked its way to one of,
if not “the” most spectacular result winning status of any sport in Aotearoa.
It amazed me the flagellation after the Olympics. To me what I saw was all
this young talent coming on with the Men’s 8 “up there” just 7 seconds off
Gold ….and the Women, “my god” I said …the women coming 4th, just
4 seconds off the winner USA.
If each one of that crew has just improved
their condition, on average through the crew, to delay lactate accumulation
at wattage (power) or speed giving a 4 seconds improvement, then we have
not just the possibility of a medal but gold.
Even though I spent most of my years with
preference in small boats, the prospect of little ol’
NZ crashing the party in the Big Boats again after all these years has this
old-oar excited beyond what any (non-rowers) could believe.
Kia kaha NZ Rowing!
women's eight crew in action in Florida Photo credit: Getty
24th January 2017
and illness or over-training syndrome.
In 1995 for the first time having on-hand
a small portable lactate testing device there was an explosion of thoughts and
enquiries on what else lactate testing could tell us apart from basic determination
of “performance at lactate threshold”.
I have forgotten who wrote that mood turns
dark a day to several days before an athlete physiologically plummets into
serious overtraining syndrome and performance capacity reversal, which as
we all know, the arresting of such performance collapse often cannot be halted
and can just carry on getting worse!.
I still wonder how many coaches and trainers
are mercilessly and uselessly thrashing athletes into poor performances?
One approach to mitigating this was exemplified
by Rushall and Pyke in “Training for Sports and
Fitness”, 1990, have athletes fill out a “Daily Analysis of Life Demands”,
which provide a “window” into the state of the athlete.
From 1995 I tested myself (relatively young
compared to now!) and several others under the duress of severe training
demands walking the tight-rope of gain or collapse as everybody felt they
A simple submax
protocol was used: 20 minute easy warmup
followed by 5 or 10 minutes at a relatively easy below La threshold pace
and lactate sample. If all was well
the HR and La at set pace would stay the same over a week or so, and then
as weeks went by reveal state of the athlete in either of 3 ways:
1. HR and La stay the same at set pace, or …
2. … they drop, hinting an improvement in
functional capacity, or …
3. … if things are either about to go bad or
have gone bad for the athlete the HR and La are higher than usual for set
The lactate sampling submax
test could be done weekly and does not stress and strain an athlete, it can
become just part of a training session!
A simpler HR version of this (without La test)
can be done everyday! …to confirm the kind of work the athlete is up for
You do not have to thrash athletes with all-out
tests to reveal that their capacities are on the improve, or in jeopardy,
or that they have stalled, hinting at it being time to review the work programme due to stagnant adaptation, or the athlete
just drifting without purpose !
An unexpectedly raised HR and La at submax can indicate another matter of concern that competes
with training adaptation and that is energy for the immune system: Our lactate meters are used in veterinary
clinics and A&E dept’s for sepsis screening: Infection raises your lactate level: A resting reading at or above what would
be your Work Threshold would be of concern!
However also, I have found some sport supplements
will (strangely!) raise your lactate level above what you would expect from
having been resting! I have not
looked for research into what you eat as negatively affecting lactate, work
capacity at La.4. If there is still
nothing on this since I looked in 1995 there is an opportunity for a
Just some observations! Hope you like and will look into it further
for yourself, to build on this!
4th April 2016.
observation on the success of NZ Olympic sports that use (or do not use)
Since first introducing convenient small portable
(hand-held) lactate testing to NZ sport in 1995 I’ve noticed that the sport
codes that involve a significant endurance component, who have used our
lactate testing product the most over the past 21 years, have been the most
Those that buy the most lactate testing strips
are hands-down unquestionably our most successful sports. Other endurance sports who one might think
would use lactate testing to be more successful are not successful.
Just a simple observation!
18th December 2015.
Maintaining Nose Breathing
and Lactate Threshold
In 1994 I came across this book by Douillard, intrigued not the least reason since it mirrored
my business’s name.
In it was his thesis on the
great benefits of nose-breathing over the all too common mouth-breathing
during exercise, or worst of all open-mouth breathing as normal while
It was not something I had thought about but
subsequently realised that I was a somewhat good nose-breather during moderate
From this point on I insisted upon it with
myself and after the period of adaptation to it, receiving clear benefits,
advised everybody who came through my door to convert to it also.
It varies between 3 weeks to 3 months for
people to adapt to strict adherence. Douillard’s thesis
is that your physiology is changed. I
thought that lactate testing should
show up some of this adaptive process.
I experimented with it in self-training
for Masters Rowing and did pretty good with
results, avoiding the frequent over-training of younger years, generating
the not too uncommon thoughts in one’s accumulated years of “what if I knew then what I know now?”
C’est la vie!
From long memory looking back, self-experimentation
with this gave equally intriguing lactate
1. In the beginning of trying this, the
upper-end of maintaining nose-breathing was a very modest pace, along with
expected Heart Rate. (Many clients
had to cut back to almost a fast-walk and could barely handle staying with
it. A lot of them arguing against it!) Lactate
at “nose-threshold” in myself then was only about
2.5 to 3 mmol. Above this intensity I would burst
into old-habit mouth-breathing.
2. As the weeks went by HR at pace dropped
and lactate went to about 3.5 mmol at max. nose-breathing
with an increased pace at this. The
HR drop at pace was not due to any increase or other factors of altered
training over that year or any other years. That is, I had not really had a drop in HR
at set paces for similar amount and level of conditioning in decades!
3. After many years from 1994 “living” this
way, the last surprise result was that I could maintain nose-breathing
(with some will - effort) at a pace-HR that equated to 4 mmol Threshold. That was some years ago now and I
remember thinking that I felt I could probably taken it up to maintain
nose-breathing at just above Threshold, say 5 mmol.
Have not to this day verified if
that is possible.
there is an experimental idea for a post-grad thesis!
There is a lot more to this but can’t put
a book here!
All the best to you for Xmas
KJ & ER Goodhew.
4th November 2015.
A relatively small amount of what is
initially high intensity tolerance work repeated daily with a largely untrained
individual has a dramatic adaptive effect on improved tolerance to that
work as measured by lactate response.
Referring to the left hand graph, there
is as said for the “tidbit” below (last week), so much in this. Edwards (1939) is another giant in early
Exercise Physiology that the profession is built on today.
“Fast forward”: Being the original
importer-distributor of small hand-held lactate testing meters in 1995, the
“Accusport” by Boehringer-Manheim, we wanted to gather as much broad
information as possible on the possible varieties of use and benefits of
such easy technology. In those days we offered the “BM&S Clinic” for personal training in any sport code,
individual or team, and a specialised weight-loss
service. Memory from those days is
that successes with people in weight-loss were at least if not more
gratifying than successes with athletes!
One client’s success that stands above all
was a 136kg woman who had “tried everything” to lose weight and
despaired. Included with
“everything” were gyms and personal trainers: Their approach was to “thrash” and basically
hurt her with relentless high intensity sessions that were unpleasant. An objective “professional” approach in
weight-loss exercise prescription simply was not there in those days.
(Being away from involvement in such services for about 12 years I have not
really looked closely at the state of the efficacy of the “weight-loss
What did we do then that was different?
We lactate tested everybody who came through our door whether their goal
was prospective Olympian, beginner runner wanting to do first marathon,
weight-loss, triple by-pass cardiac rehab, wanting to look good for her
wedding, a horse, …and even the postman! True!
When my favourite client story first came
through the door she was scared of exercise. I told her that she would lose
weight with walking at correct intensity and that it would be easy compared
to what she had done and that we would work it out by testing her blood to
make sure the exercise is not at unpleasant and unnecessary high intensity
which puts damaging acid into your blood and through your body. That is,
putting things into simple terms without off-putting sport science jargon.
As a prelim., with HR monitor on we set
off up the road which leads to Frasers Gully about 2.4 km in length with a
more challenging slope in its last ¼, but which is in the most part a
barely perceivable gradual slope to find that she could not walk around
100m without bursting into rapid mouth breathing and appearing
bothered. Next time I conducted a
necessarily “off-the-cuff” modified lactate assessment protocol just to
find where her 4 to 5 mmol HR intensity was.
Several times per week we met to do
Frasers Gully interval style whereby as soon as her HR climbed above her
original 5mmol HR we would stop, let it drop for a minute or two and then
on until the alarm called for the next stop.
At the end of just one month my 136kg
client who had lost about 6kg walked to the top of the gully without one
stop or alarm of high HR. Her blood
pressure, borderline before, was now normal.
She told me she was “…amazed how easy and
pleasant the exercise intensity was…” for her to achieve so much.
The graph on the left in today’s copy
& paste reminded me of the mechanism of this favourite client success.
23rd October 2015. : FYI: If
you thought I was talking crazier than usual, some kind of load-up error
occurred where the wrong table appeared! Corrected 25/1/2017
Classic tabulated data on responses to
various brief training impulse/recovery cycles from the now classic
eponymous text by Astrand and Rodahl,
of Work Physiology”.
Yes! I still have my copy from undergrad
days! In it is this referenced
original study by Christensen et al, 1960!
I’ve added to the right an approx.
conversion of mg/100ml to mMol.
There is so much that can be taken from this
table that I cannot really get into too much here since having looked at it
and making use of it in training individuals years ago. In past days “in da game” I got a
reputation for taking what people perceived as quite ordinary ranked
athletes to a level unexpected. One
day watching an athlete come in a spectator said to me, “…how is that
possible”? I was young then, saying somewhat flippantly, “training
secrets”. This was later not taken well being accused of offering
unfair advantage to the athlete over others in the club who were expected
to dominate in a predicted procession of race positions. Envy amongst
coaches in NZ sport is a not uncommon terrible thing; I did not last long
in that club!
How did I use this table? The issue for the average athlete is that
they have average aerobic capacity, average VO2max, and come up against
gifted athletes with lungs and heart of a horse. What is one to do? We know VO2 is trainable to a significant
degree, but more so is % VO2 that AT kicks off a rapid lactate
accumulation as determinant of endurance performance. All we can do is optimize the average
athlete’s VO2 and then shift their “threshold” as close as possible toward
their VO2max. (…plus effective race
strategy, work on motivation, psychology, and structural soundness –
anatomy – for freedom from potential to injury. The latter most important
since however you do it, VO2max pace work is high stress work. I don’t
advocate you do any of this with a beginner!)
We also know that anaerobic-lactate work
destroys aerobic enzymes for upto several days and opens one up to illness
(immune compromise). You can see
from the table that doing VO2max pace continuously for 4 minutes takes
lactate to 16.7 mMol and therefore aerobic
enzymes destroyed. This is not suitable training! Coaches who over max test or over-race
their athletes in this form are simply stuffing them up!
You can see for an already fairly well
trained athlete work/rest intervals of 10:5 and 15:10 elicit too high
lactate response, destroying aerobic capacity for subsequent days.
You can see the best strategy to elicit a
near maximum VO2 of 5.3 with a lactate response equivalent to “going for a
jog” is 15:15.
Experiment with this and during it wack in
a lactate test or two to determine your athlete’s individual response to
I can assure you this works a treat. I self-tested (when I was young
with “OCD” for discerning “secrets”) and used it on numerous athletes to
raise them up quickly. If you
schedule it weekly use their mood to decide whether to stick with it
religiously as per schedule or not.
Go to Archive of Lactate testing “tidbits”: Click!
Link will be live soon!
Do you have a story for us, maybe research you are
doing? We love to hear and share your stories.
Comparing the old with the new!
NOTE, LP(1) Meters and strips no longer
or Team Trainer/Coach use
and health prescription (Green prescription)
faster, takes just 15 sec’s
smaller sample required
& Software available, connectivity to PC.
“The original” (1998)
- Oldie but a
Ø Child-birth – foetal
hypoxia screening – Maternity Wards NZ DHB’s
- Sepsis screening
Sport Science/Medicine; Personal or Team Trainer/Coach; Olympic sport teams
and health prescription (Green prescription)
Currently LP (1) test strips remain
available until further product development!
Monday, 23rd January 2017
trend since Rio carries on!
50% more Meters than we ever have with 2/3 gone either before they got here
or within 2 weeks. Amazing! At current trend we are going to run out
before next shipment gets in, don’t procrastinate!
confidential deal on sets for educational exercise physiology (or other)
labs is available: Minimum quantity
= 5 Meters.
minimum quantity was chosen since it could meet the small number of
students in labs of size 10 to 15 students with one each of tester, recorder,
Secondary “Sport Science” students could do this with tester, recorder, and
roping in a subject from say the school rowing squad!
Just a personal view:
All those in their last year of school rowing wanting to maintain their interest
and progress in the sport would have their decision making greatly helped
with a clear lactate profile revealing performance improvement progress and
potential trajectory which is much better shown with a lactate profile
rather than all-out Erg tests which can wildly disguise capacities and
potential for improvement!
us for the Educational Quantity Deal
on 5 (or more!):
Enquire now: Email (click here!)
Friday, 9th December 2016
the Rio Olympics NZ Sport has gone to another level!
A notable phenomenon has occurred since the Rio
most of the small portable lactate testing strips in NZ, for a long time now,
being since the technology was first launched and we picked it up that same
year in 1995: This has afforded us a unique position of insight into who is
doing what in terms of science based training in Aotearoa, particularly in
thing that we noticed by the millennium was that the sport that does the
most lactate testing is our most successful Olympic sport, hands-down, and
is still in that same position doing 21 years of regular systematic lactate
assessments in NZ! An immense
knowledge base is built up with all that time. It has had us asking what the other NZ endurance sports are
doing for sport science. The results seem very telling to us. But, to be
direct in asking about that is not a comfortable role or position for us as
since Rio there has occurred a phenomenon that may signal a dramatic change
to this status quo:
each Olympics since Atlanta, USA, 1996, after these events we would endure
a great drop off in demand of strips and Meters …
Little to no lactate testing would be done post Olympics
year after Rio, the expected high
demand of pre-Olympics has been superseded by higher demand AFTER
never had anything like it in 21 years and we’re going to have a job
getting our next shipment here before we run out, since projections are to
have us run out 5 months ahead of schedule!!! We’ve got demand wrong by a month over
the years but never by 5 months!
wondering if there has been a spark of belief occur at Rio that is going to
take NZ to a new level at the next Olympics?
“we” thrown off our seeming forever cringing excuse of “small nation”, with
often taking the choker role at the big-time, with FINALLY NOW HAVING AN
INFECTIOUS BELIEF IN OURSELVES that any athlete lining up beside us from
anywhere in the world is just another athlete, and that our young talent
are just going to go for it like never before?
we really are great at this thing called the Olympics?
know! Anyone else have any ideas why
Trainers / Sp. Scientists are right now going for it like never before?
Keen to hear.
it deems great for an exciting future.
Monday, 4th April 2016
We are still looking for a few more “On-sellers” (retailers) in various
areas around NZ: Contact us:
Wednesday, 19th February 2014
concentration on his way to winning Taranaki Triathlon several years ago…
…uses LP2 to good effect.
Approximately 12 months ago I started
using my Lactate Pro 2 Meter for Training and Racing purposes. Previous to
this I relied heavily upon Heart-rate Monitors and “subjective feel” data to
ascertain my performances and success/failures.
The most crucial benefit I find is
that it allows me to train and race way more precisely and accurately. It
gives me the added advantage of knowing how to increase my Lactate Tolerances
when in heavy/extended sessions or even when doing a recovery type mode
work-out. I can therefore correlate my Heart-rate a lot closer to my Lactate
Thresholds, thus allowing me to race smarter, quicker, efficiently and
When racing, it’s critical not to
start at to higher Heart-rate and Lactate level, as this generates into
fatigue, poor performance and the inevitable bonking. By measuring and
testing your Lactate levels in training, you are then able to maximise your
performance and results, come race day. It’s such a valuable and necessary
tool in my training and racing programme, that without it, I would be always
wondering and 2nd guessing at why my performances were not at their highest
level. One thing that is for sure – Blood Lactate levels DO NOT lie!!, even though you may think and feel differently.
The Lactate Pro 2 gives you that
complete and 100% answer in how to improve your training speeds, conditions,
results and performances, via the easy and simple testing methods, plus the
added benefits of being simple to test, quick on results, with accuracy and
lightweight to use/carry.
I thoroughly recommend the Lactate
Pro2 Meter as the best, overall piece of training equipment that has lifted
my performances, and allowed me to compete at my top potential. As a serious
Age Group Triathlete, the Lactate Pro 2 has given me an added advantage to
optimise my full athletic potential, and permits me to train / race in the
best possible shape and condition.
Mens 60 - 64 Age Group, Tri New
Compare the *New* with the Old
period of the test strips
a calibration strip
calibration (No coding)
In Summary: The new LP2 is 75% faster at 15 seconds,
with a lower and higher lactate value measurement range, and increased
temperature use range down to 5C. Its calibration is automatic, no longer
requiring a calibration strip. At 18 months from date of manufacture the test
strips have a 50% longer life before expiry date. The LP2 can take 330 test
results in memory which will be downloadable for analysis on PC software by
cable. Software & cable availability at this date yet to be
announced. The Meter can host upto 3
user identities: This could work out quite economical for a small group
owning a Meter between them.
Frequently Asked Questions & Answers!
Meter production has stopped, will they stop producing the LP(1) test strips
and when? We
don’t know the answer to this! Best you
It stands to reason that
the manufacturer is giving you (…all LP users) a fair transition time to
upgrade by purchasing a new LP2 Meter FROM JUNE this year! You’ve already had 6 months to consider
upgrading. If you leave it until a halt to production notice is given, and
join those who panic to get one due to not being organised, then you could
find we may not be able to supply you immediately with a Meter for some time
when you urgently want it, joining those ranks that would cause a scarcity of
supply through sudden world-wide demand!
This “segways” (segues) to:
Understand that when we
place our orders with the factory we must predict 4 to 6 months ahead as to
what we think the demand will be for any given month. This is especially so with consumables.
Most people want maximum duration to expiry, therefore we do not order in by
excessive quantities for the chance to cover some exceptional (unpredictable)
high demand scenarios as customers do not want stock that has been shelved
too long. We can of course predict high demand for Olympic Year! If we are hit with sudden demand above
projections or the manufacturer is hit by huge demand world-wide then it is
possible that we are unable to supply you for a month or more. Therefore, You should always plan to order
a minimum one month ahead of important testing schedules to avoid the 0.1%
chance we could fail to supply overnight for your order. And, you should also employ some basic
management yourself by setting a minimum quantity re-order
trigger strategy for test strips so that you do not completely run out!
Can I use the new LP2 strips in my old
Meter or LP(1) strips in new LP2 Meter? No!
Can you take the
steering-wheel out of your wonderful old 1956 Volkswagen Beetle and put it in
your brand new 2012 Series-5 BMW? See
“Compare” above: The LP(1) test strip measuring time
is 60 sec’s, the LP2 15 sec’s; How would a 15 sec chemical reagent
measuring-time strip work in a 60 sec Meter?
The LP2 sample is just 0.3 ul, the LP(1)
needs almost twice as much sample volume; If you could get it in the machine
the LP1 Meter would read that half volume as an “error”! Calibration: The LP2 requires no calibration-strip checking,
this is now automatic. If you tried to put a non-automatic calibrating LP(1) strip into an LP2 Meter how would the Meter read
it? And… …you
got it already!
Is the Software and cable included? No!
The new LP2 Meter and
test strips are at exactly the same cost price-point from the manufacturer as
the original LP(1) Meter and strips. We think this is an excellent deal for you
in an upgraded product that incorporates multiple superior features. The LP(1) did not
have software and connecting cable, which is a significant material addition
to the original product, and is therefore of course an item which is an
add-on sale to your LP2 purchase.
I wish to purchase the LP2 Software
& Connector. We will notify here when these are
available. We do not have a date yet that these will be shipped to us.
What reliability and validation
research do you have? …is it more accurate than the LP(1)? We have no product comparative data yet.
The product is brand
new! We may have been the first
country outside of Japan to stock and supply it.
Apart from Australia I do
not know any other country with it, it is that new. We get purchase requests
from Germany, South Africa, USA …!
I am only speculating
that the product was developed and validated entirely in-house, perhaps in
co-operation with Universities of Japan.
Would it not stand to reason that a new upgrade model would be more
reliable, accurate, and fully validated over the original before a factory
would be tooled up with tens of million$ expense and before doing production
runs of it? Take my word; from my
experience with this Japanese company since 1997, you can be confident in
Arkray LactatePro2 as being a superior product.
It is wide-open for you to make a name for yourself by doing some product comparison
studies right now and being first in the world to publish on it! Come on “kiwi” get on with it!
Here (NOW!) Be the first to get one.
We already have advance orders:
Order yours NOW! 1/6/2012
Arkray Website News Release
The smallest meter in its class just
got easier to use. Even better performance for ARKRAY’s card-sized blood
lactate measurement device
Inc. (Headquarters: Kyoto City, Nakagyo-ward), a manufacturer of sample
test devices and in vitro diagnostic reagents is set to launch its
new and simple blood lactate meter, the Lactate
Pro™ 2 LT-1730 on 17 April. In its card size form, it is small
enough to allow measurement anywhere and boasts increased performance for
the rapid measurement of lactate.
in blood is an essential marker in the fields of sports physiology, sports
science and training. The simple and rapid testing of lactate is a big
benefit to both research and medicine.
answer the needs of the market, ARKRAY released the Lactate Pro TM back in
February 1997, which marked a significant reduction in size for blood lactate
at the time. This new card sized device enabled simple measurement of lactate
anywhere and helped to support clinical testing across many different
is now set to release the simple blood lactate meter, the Lactate Pro™ 2 LT-1730 together with its compatible reagent/
measurement electrode, the Lactate
Pro™ 2 Sensor. The measurement time has been cut
by 75%* to just 15 seconds; the minimum sample volume reduced by over 90%* to
just 0.3µL and the basic performance of the device improved significantly.
The hassle associated with correction of reagent lots has been eliminated
which also helps to reduce the risk from correction errors.
will continue to answer diverse testing needs in the market.
comparison to existing ARKRAY products:
Lactate Pro™ 2
Pro™ 2 Sensor
Main features of the Lactate Pro™ 2
●A simple blood lactate device, the smallest of its kind
Palm-sized for measurement in any environment: now you can measure lactate
●Minimum sample size: just 0.3µL
Development of a new reagent sensor: measurement possible with just 1/10th
the volume previously needed
●Measurement time: 15 secs
On site measurement is possible using finger-stick blood: results are shown
in just 15 seconds from application of blood to the sensor- one quarter the
time needed for measurement with previous devices.
●No correction needed
No more hassle from correction of sensor lots (insertion of sensor chips
before measurement): this also helps to reduce the risk from correction
Blood lactate is often used in the scientific training for endurance sports
such as soccer, swimming and athletics. As the intensity of the exercise
increases, so too does the concentration of lactate in the blood. By
measuring this change, training can be configured to delay sharp rises in
blood lactate. (There are also Medical
and Equine/Veterinary applications – KJG, BM&S Imports-NZ.)
Lactate Analyzer Lactate Pro™ 2 LT-1730
17 April 2012 (Tues.)
Lactate in blood
LOD enzyme electrode method
15 sec/ sample
Lactate Pro™ 2 Sensor (Electrode for measuring lactate in blood)
Auto-correction using internal temperature sensor
Temp: 5-40 C, Humidity 20-80% RH (No condensation)
3V lithium battery/ CR2032 X1
50mm(W) X 12mm(D) X 100mm(H)
Approx. 45g (Incl. batteries)
Do you need an
“ordinary” Bathroom scale or a Kitchen scale?
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world’s first small hand held lactate test meter we have been FIRST in NZ to
supply you with the best.
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(ensuring correct exchange rate, shipping and GST have been accounted
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research dept’s (e.g. Lactate testing of salmon!)
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This article posted here 26 Nov. 08 cited from HIV InSite,
University of California, San Francisco.
lactate testing predicts mortality of severe sepsis in a predominantly HIV
type 1-infected patient population in Uganda
UCSF Institute for Global Health Literature Digest
Published April 17, 2008
Moore CC, Jacob ST, Pinkerton R, Meya DB, Mayanja-Kizza, Reynolds SJ, et al.
lactate testing predicts mortality of severe sepsis in a predominantly HIV
type 1-infected patient population in Uganda. Clin
Infect Dis 2008 Jan 15; 46(2):215-22.
To evaluate the ability of a handheld portable whole-blood lactate (PWBL) analyzer to predict mortality in
patients who are admitted to the hospital with severe sepsis.
A prospective observational study.
An accident and emergency department of Mulago
Hospital, a national referral hospital in Kampala, Uganda.
72 patients were enrolled in the study. Inclusion criteria were ≥18
years of age and admission to a medical ward, along with: 1. two or more
systemic inflammatory response syndrome criteria (body temperature, >38°C
or <36°C; heart rate, >90 beats/min; respiratory rate, >20
breaths/min; or peripheral WBC concentration, >12,000 cells/mm3 or
<4000 cells/mm3; or thermodysregulation; 2.
systolic blood pressure ≤100 mm Hg; and 3. a suspected infection. Exclusion
criteria included acute cerebrovascular events, gastrointestinal hemorrhage, or admission to the surgical or obstetrics
and gynecology ward.
There was no intervention in this study. This analysis was conducted among a
subset of 253 patients recruited to study the incidence, management, and
outcomes of sepsis. From this sample, the first and last 50 consecutively
enrolled patients were recruited to determine the predictive value of PWBL in
predicting mortality from sepsis. Background information - including age,
sex, HIV-1 serostatus, and prescribed
antiretroviral medicines (ARVs)-was recorded. At patient enrollment,
temperature, heart rate, respiratory rate, and blood pressure were measured.
To determine outpatient survival, an attempt was made to telephone patients
30 days after their discharge from the hospital. A rapid HIV-1 test and
malaria smear were performed at Mulago Hospital. A
local private clinical laboratory provided results of lactate and bicarbonate
analysis. PWBL was obtained using a lancet to collect a drop of whole blood
from the patient's finger, for analysis by a handheld portable device. This
instrument uses enzymatic determination and reflectance photometry of lactate
in the plasma portion of whole blood using a measurement strip. Standard
laboratory serum lactate (SLSL) concentration was obtained by phlebotomy for
venous blood samples. Within 2 hours of sample collection, the sample was
transported in a standard serum tube via a cooler to the clinical laboratory,
where blood was centrifuged and serum was removed for use in the lactate
Information was available for 72 of the 100 enrolled subjects. The mean age
of participants was 35.7, 61.1% were women, and
81.9% were HIV infected, with a mean CD4 lymphocyte count of 88.6 cells/ mm3.
These 72 patients were similar to the larger study population in age (mean
age, 35.7 vs. 33.8 years), sex (61.1% vs. 59.1% female), HIV-1 seropositivity (81.9% vs. 86.6%), and ARV status (13.9%
vs. 10.6% ARVs prescribed). Fifty-nine (81.9%) of 72 evaluated patients were
infected with HIV-1. The in-hospital mortality rate was 25.7% (18 of 70), and
the in- and outpatient mortality at 30 days was 41.6% (30 of 72). PWBL was
positively associated with in-hospital but not outpatient mortality (p
<.001). The receiver operating characteristic (ROC) area under the curve
for PWBL was 0.81 (p <.001). The optimal PWBL concentration for predicting
in-hospital mortality (sensitivity, 88.3%; specificity, 71.2%) was ≥4.0
mmol/L. Patients with a PWBL concentration ≥4.0
mmol/L died while in the hospital substantially
more often (50.0%) than did those with a PWBL concentration <4.0 mmol/L (7.5%) (odds ratio, 12.3;
95% confidence interval, 3.5-48.9; p <0.001). SLSL levels were lower among
survivors than among deceased. The ROC under the curve for predicting
in-hospital mortality was 0.72 (p=0.004). SLSL results were inconsistent and
less predictive of mortality than were those of PWBL.
The authors conclude that PWBL testing can quickly identify patients who
require immediate interventions, and it should be included in
evaluation and treatment algorithms for septic patients. PWBL testing could
be used in village health posts, for earlier transfer of septic patients to
facilities with a higher level of care, and in referral hospitals, for triage
of patients to acute care settings where appropriate resuscitation can begin.
Elevated lactate concentrations and poor clearance of lactic acid are known
to increase mortality from severe sepsis. Lactate concentrations, combined
with other laboratory measures, are used to guide early management of sepsis.
Measurement of lactate concentration in developing countries is difficult
because of limited resources. An effective, inexpensive method to measure
lactate concentration without sophisticated laboratory resources has the
potential to offer a method to identify patients in greatest need of rapid,
aggressive treatment of sepsis.
The use of the PWBL and its value in predicting mortality from sepsis in
populations with high prevalence of HIV has potential to assist health care
workers in resource limited settings in prioritizing these patients.
Hospitalization, availability of support for potential multisystem failure,
and rapid administration of antimicrobial agents can be delivered more
urgently in persons with high lactate concentrations.
This article posted here 26 Nov. 08
cited from Lab Tests On-line.
Testing in Acute Assessement
What is being
This test measures the amount of lactate in the blood or, more rarely, in the
fluid. Lactate is the ionic (electrically charged) form of lactic acid.
It is produced by muscle cells, red blood cells, brain, and other tissues
energy production and is usually present in low levels in the blood. Aerobic
energy production is the body’s preferred process, but it requires an
adequate supply of oxygen. Aerobic energy production occurs in the
mitochondria, tiny power stations inside each cell of the body that use
glucose and oxygen to produce ATP (adenosine triphosphate), the body’s
primary source of energy.
When cellular oxygen levels are decreased,
however, and/or the mitochondria are not functioning properly, the body must
turn to less efficient anaerobic energy production to metabolize glucose and
produce ATP. In this process, the primary byproduct
is lactic acid, which can build up faster than the liver can break it down.
When lactic acid levels increase significantly in the blood, the affected
person is said to have first hyperlactatemia and then lactic acidosis (LA).
The body can often compensate for the effects of hyperlactatemia, but LA can
be severe enough to disrupt a person’s acid/base
(pH) balance and cause symptoms such as muscular weakness, rapid
breathing, nausea, vomiting, sweating, and even coma.
Lactic acidosis is separated into two types: A and B. Type A may be due to inadequate oxygen uptake in
the lungs and/or to decreased blood flow (hypoperfusion)
resulting in decreased transport of oxygen to the tissues. The most common
reason for this is shock from a variety of causes including trauma and blood
loss, but LA may also be due to conditions such as heart
heart failure, and pulmonary edema (fluid in
the lungs). Type B is caused by conditions that increase the amount of
lactate/lactic acid in the blood but are not related to a decreased
availability of oxygen. This includes liver
storage diseases (such as glucose-6-phosphatase deficiency), drugs and
toxins, severe infections (both systemic sepsis
and a variety of inherited metabolic and mitochondrial diseases (forms of
muscular dystrophy that affect normal ATP production). Strenuous exercise can
also result in increased blood levels of lactate.
Testing in General Practise
With the advent
of the world’s first small hand-held lactate test meter launched in 1994 by
Boehringer Mannheim, the use of lactate prescription for effective and
objective “Green Prescription” was already well considered. My personal observation is that the Europeans
are well ahead of us in this medically correct and safety orientated approach
to intensity prescription of exercise. It is this correct medical approach that
has been begging for implementation here in NZ. Fourteen years later (2008)
the right Doctor with knowledge & motivation to champion this cause here
is yet to be forthcoming!
view larger image in viewer on your PC click here.
2. This article posted here 26 Nov. 08 cited on-line at Wiley Interscience.
Interval versus continuous training in patients with severe
COPD: a randomized clinical trial Eur Respir J 1999; 14: 258–263.
R. Coppoolse*, A.M.W.j. Schols*, E.m. Baarends*, R. Mostert**, M.a. Akkermans**, P.p. Janssen**, E.F.m.
**Astmacentre Hornerheide, and *Dept of Pulmonology,
Maastricht University, the Netherlands.
Correspondence: A.M.W.J. Schols
Dept of Pulmonology
P.O. Box 5800
6202 AZ Maastricht
Fax: 31 433875051
information is available regarding the physiological responses to different
types of exercise training in patients with severe chronic obstructive
pulmonary disease (COPD). The aim of this study was two fold: firstly, to
investigate the physiological response to training at 60% of achieved peak
load in patients with severe COPD; and secondly to study the effects of
interval (I) versus continuous (C) training in these patients.
patients with COPD (mean±sd
forced expiratory volume in one second: 37±15% of predicted, normoxaemic at rest) were evaluated at baseline and after
8 weeks' training. Patients were randomly allocated to either I or C
training. The training was performed on a cycle ergometer, 5 days a week, 30
min daily. The total work load was the same for both training programmes.
resulted in a significant increase in oxygen consumption (V 'O2)
(17%, p<0.05) and a decrease in minute ventilation (V 'E)/V 'O2
(p<0.01) and V 'E/carbon dioxide production (V 'CO2)
(p<0.05) at peak exercise capacity, while no changes in these measures were
observed after interval training. During submaximal exercise a significant
decrease was observed in lactic acid production, being most pronounced in the
C-trained group (-31%, p<0.01 versus -20%, p<0.05). Only in the
I-trained group did a significant increase in peak work load (17%, p<0.05)
and a decrease in leg pain (p<0.05) occur. Training did not result in a
significant improvement in lung function, but maximal inspiratory mouth
pressure increased in both groups by 10% (C: p<0.05) and 23% (I: p<0.01).
The present study
shows a different physiological response pattern to interval or continuous
training in chronic obstruction pulmonary disease, which might be a
reflection of specific training effects in either oxidative or glycolytic
muscle metabolic pathways. Further work is required to determine the role of
the different exercise programmes and the particular category of patients for
whom this might be beneficial.
Equine Trainer & Veterinary
Davie is probably the pre-eminent advocate in Australasia of effective
training of race horses incorporating lactate testing. The great value of his book is the
simplicity of its explanation. Anybody
can use it to catapult the training of their horses into 21st
Century method to earn positive performance improvements.
Price NZ$ relative to currency x-rate!
Bad news for heavy riders and narrow
March 3, 2008
Researchers in the US have bad news for
overweight horse riders. A study has found that horses that have to carry
between 25 and 30 per cent of their bodyweight have more physical problems
related to exercise than those who carry 20 percent or less.
Horses carrying 30% body weight showed a
significant increase in muscle soreness and muscle tightness scores. The
changes were less marked when they carried 25% body weight.
Dr Debra Powell .... conducted a study ....performing a standardised ridden
exercise test in an indoor school arena. After five minutes active walk to warm
up, the horses were ridden at a trot (3m/s) for 4.8km, followed by 1.6km at a
canter (5m/s). This exercise schedule was chosen to simulate a 45-minute work
period of work typical of an intermediate-level riding school horse.
The researchers measured heart rate, plasma lactate concentration and creatine
kinase. Lactate is produced in the muscles during exercise. At low levels of
work the body can metabolise it and so levels in the plasma remain low. As
the work level increases the rate of lactate production exceeds the body's
ability to remove it and so concentrations rise. Creatine
kinase (CK), an enzyme present in the muscles, is released into the blood as
a result of some types of muscle damage.
... Plasma lactate levels were higher
immediately after exercise and 10 minutes after end of exercise, in horses
that carried 30% of their body weight....
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