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Important Notices:

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Tuesday, 17th April 2018

 

 

 

Shipping News!

 

Shipment is in!

 

Made great time. Your backorder already on way to you, there tomorrow, unless your address is RD where it’ll be the usual extra day.  

 

Happy testing!

;-)

 

KJG

 

 

 

 

Friday, 13th April 2018

 

 

 

Shipping News !

 

Shipment has left the factory:

 

ETA Wednesday 18th

-  estimated! -  

 

Backorders to go out first, of course!

 

Any “PBD” orders received to 5 pm Tuesday 17th will join priority backorders list treated with utmost urgency.

 

Orders received after Tuesday will likely not be actioned until following Monday, 23rd April.

 

PLEASE stock up from this shipment early even if you don’t need them for quite a while since a lot of it has already gone, pre-sold, with the next shipment over a month away, we will likely run out again!

 

 

 

 

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Update: Friday, 13th April 2018.

Monday, 19th February 2018

 

 

 

NZ Masters Games Indoor Rowing!

 

I will put a wee lactate-testing story here soon, 20 years on from my last go at racing on “the machine”!

 

In the meantime check out my lactate profile building up for NZ Masters Games scrolling down here… http://www.tanita-nz.co.nz/My%20TANITA%20testimonials%20071117.htm#KJG

 

As at 13/4 I am still to post an update of season end results … soon I promise! …biz has been flat-out, not much time to waffle on! ;-)

 

Regards

KJG

  

 

 

Friday, 8th December 2017

 

 

Rowing again!

 

Again, apology for my sport bias but it is the one I am most familiar with from my youth and the principles are applicable to many sports!

 

“Power-2-Weight” :  PART ONE

 

When I came out of Phys Ed School in ‘83 I went back to rowing a couple years later to experiment on myself with what I’d learnt.  On arriving, University “Easter Tourney” rowing was not what it is today, feeling it mickey-mouse, so I didn’t bother with it for 4 years, proving costly when trying to get “back on the oar” with any kind of representative ambitions.  Was a so-called adult student going in, coming back to career hunting and rowing at 26.

 

I became increasingly fascinated with the concept of Power-to-Weight as a determinant of performance.  Soundly frequently defeated by about half a dozen of the countries top Light-weights (<72.5 kg), being only a “medium-weight” myself with a best racing (and health) weight of about 82kg, I had to also contend with about a dozen ahead of me in the Heavy-weight Single Scull category.  Did okay with placings in Intermediate and Senior events.

 

Noticed improved condition through a Season coincided with becoming very trim with reduced weight (from around an off-season 88kg, back in the day when there was such as off-season!).  Was performance improvement just a factor of improved cardio-vascular function or, and, if both, was it about carrying less weight? … which had the most impact?

 

Then there was the phenomenon of Philippa Baker and Brenda Lawson “pocket rockets” who for years annihilated any truly heavy weight women at the NZ Champs and won individual world titles in Light Weight and Under 23 respectively, and then took on the world together reigning for several years in the Double Sculls.

 

It was their performance that inspired me to create a power-to-weight performance table that gave predictive on-water single scull times … (requested data for only on “even” courses, meaning no tail-wind or current) … which was taken from one’s bodyweight and 2000m Ergometer score.  I put a survey to NZ’s top rowers who kindly allowed use of their data (which could not personally identify them anyway) and using mine and other modest performers the spread on the table from average to superior was completed. It was developed on old Concept2 B Models. I suspect the algorithm has changed somewhat from those days so that for such a performance predictive table to be used today it would need to be re-done for today’s “E” model and superior technology in faster carbon-fibre skiffs. We were only just transitioning from wooden oars in my-day!

 

The impressive German immigrant Herman Krutzman residing in Cambridge, himself an accomplished sport scientist, Olympic level coach, and builder of skiffs branded in his own name, Krutzman, was an incredible source of additional “real” education for this “newbie” in hydrodynamics revealing the massive increase in resistance (additional force required from what is a modest “biological engine”) for the increased surface area of a skiff due to increased weight.

 

If a variety of people today sent me their best Single race time (again, even conditions only), with their Erg 2K PB, and their bodyweight for their on-water performance I could modernize this predictive table.  I have used it for good motivational effect when I was coaching about 20 years ago!

 

If you want the old table as it is, I will see if I can dig it out, drop me an email, or if you have a collection of old NZ Rowing mags it appeared there around the late ‘80’s (think it was!).

 

Then there was the spectator confusing sensational Cohen and Sullivan, multi-World and 2012 Olympic Double Scull Champions, medium-weights and average height (just like me!) that resumed an interest in this Power-2-Weight thesis.  When they stood on the dais against those they had defeated they appeared “little guys” at around 6 foot!  They also paced their races in a style which was uncommon (subject for another “lactate tidbit” article one day!). They would appear to sit modestly in the field to finish over the last 500m with a blistering sprint that put fans on the edge of seats, exhilarating, as they carved through the field.

 

This “Power-2-Weight” subject had its original roots way back to a particular graph in the now classic undergrad “Textbook of Work Physiology” by the legends Astrand and Rodahl (2nd Ed, 1977) that lends to this (above) “story” to this day.  

 

 

 

Traditional graphs of VO2max show that the heavier you are the greater likelihood that you have the highest VO2max. However, if you put a spread of people from those with light bodyweight and relatively low VO2max against heavier people with higher VO2max you do not necessarily have a predictor of performance in times, or across a finish line, in cycling, running, or on water events!

 

However!  The lower graph shows you that you do have a predictor of performance if you correct for bodyweight by a function of its -2/3.

 

This means that instead of the all too common standard expressions of simple litres per minute VO2max or milliliters per minute per kilogram, use milliliters per minute per kilogram to -2/3.

 

Simply, it evens out (or accounts) for the effect of bodyweight on VO2max leaving you an assessment of just who has the better VO2max irrespective bodyweight!

 

Why this third formulation is not used as one standard in High Performance sport I have no idea, maybe someone can tell me. (Maybe I’ve been away from ivory towers too long!  ;-) It has only been around as a clearer predictor than straight VO2max or milliliters per kilogram since the leading-light sport scientists of the ‘60’s!  Sure, Lactate Threshold is a better “fitness” indicator, but VO2max assessment still has the place for predicting the ultimate performance capacity and international competitive potential (rank) of an individual.

 

So what practically is the value of all this long commentary for you? How do you use this to improve performance?

 

That is the subject for Part Two!  

 

Will try and get onto this before or over the Xmas break.  If you don’t hear from me before, or I you, YOU HAVE A GREAT XMAS!

 

;-)

 

KJG

 

KJ & ER Goodhew

BM&S Imports – lactate.co.nz   

 

 

 

 

 

Thursday, 28th September 2017

 

 

 

Rowing!

 

Please excuse this writer’s bias …an old rower!

 

NZ Rowing has hard-worked its way to one of, if not “the” most spectacular result winning status of any sport in Aotearoa. It amazed me the flagellation after the Olympics. To me what I saw was all this young talent coming on with the Men’s 8 “up there” just 7 seconds off Gold ….and the Women, “my god” I said …the women coming 4th, just 4 seconds off the winner USA.  

 

If each one of that crew has just improved their condition, on average through the crew, to delay lactate accumulation at wattage (power) or speed giving a 4 seconds improvement, then we have not just the possibility of a medal but gold.

 

Even though I spent most of my years with preference in small boats, the prospect of little ol’ NZ crashing the party in the Big Boats again after all these years has this old-oar excited beyond what any (non-rowers) could believe.

 

Kia kaha NZ Rowing!

 

KJG  

The women's eight crew in action in Florida Photo credit: Getty

 

 

Tuesday, 24th January 2017

 

 

 

Lactate testing and illness or over-training syndrome.

 

In 1995 for the first time having on-hand a small portable lactate testing device there was an explosion of thoughts and enquiries on what else lactate testing could tell us apart from basic determination of “performance at lactate threshold”.  

 

I have forgotten who wrote that mood turns dark a day to several days before an athlete physiologically plummets into serious overtraining syndrome and performance capacity reversal, which as we all know, the arresting of such performance collapse often cannot be halted and can just carry on getting worse!.

 

I still wonder how many coaches and trainers are mercilessly and uselessly thrashing athletes into poor performances?

 

One approach to mitigating this was exemplified by Rushall and Pyke in “Training for Sports and Fitness”, 1990, have athletes fill out a “Daily Analysis of Life Demands”, which provide a “window” into the state of the athlete.

 

From 1995 I tested myself (relatively young compared to now!) and several others under the duress of severe training demands walking the tight-rope of gain or collapse as everybody felt they must do:

 

A simple submax protocol was used:  20 minute easy warmup followed by 5 or 10 minutes at a relatively easy below La threshold pace and lactate sample.  If all was well the HR and La at set pace would stay the same over a week or so, and then as weeks went by reveal state of the athlete in either of 3 ways:  

1.  HR and La stay the same at set pace, or …

2.  … they drop, hinting an improvement in functional capacity, or …

3.  … if things are either about to go bad or have gone bad for the athlete the HR and La are higher than usual for set effort.

 

The lactate sampling submax test could be done weekly and does not stress and strain an athlete, it can become just part of a training session!  

 

A simpler HR version of this (without La test) can be done everyday! …to confirm the kind of work the athlete is up for that day.

 

You do not have to thrash athletes with all-out tests to reveal that their capacities are on the improve, or in jeopardy, or that they have stalled, hinting at it being time to review the work programme due to stagnant adaptation, or the athlete just drifting without purpose !

 

An unexpectedly raised HR and La at submax can indicate another matter of concern that competes with training adaptation and that is energy for the immune system:  Our lactate meters are used in veterinary clinics and A&E dept’s for sepsis screening:  Infection raises your lactate level:  A resting reading at or above what would be your Work Threshold would be of concern!

 

However also, I have found some sport supplements will (strangely!) raise your lactate level above what you would expect from having been resting!  I have not looked for research into what you eat as negatively affecting lactate, work capacity at La.4.  If there is still nothing on this since I looked in 1995 there is an opportunity for a student!

 

Just some observations!  Hope you like and will look into it further for yourself, to build on this!

 

KJ Goodhew.

 

 

 

Monday, 4th April 2016.

 

 

 

A correlative observation on the success of NZ Olympic sports that use (or do not use) lactate testing.

 

Since first introducing convenient small portable (hand-held) lactate testing to NZ sport in 1995 I’ve noticed that the sport codes that involve a significant endurance component, who have used our lactate testing product the most over the past 21 years, have been the most successful.  

 

Those that buy the most lactate testing strips are hands-down unquestionably our most successful sports.  Other endurance sports who one might think would use lactate testing to be more successful are not successful.

 

Just a simple observation!

 

KJ Goodhew.

 

 

 

 

 

 

 

Friday, 18th December 2015.

 

 

Maintaining Nose Breathing

and Lactate Threshold

In 1994 I came across this book by Douillard, intrigued not the least reason since it mirrored my business’s name.

 

In it was his thesis on the great benefits of nose-breathing over the all too common mouth-breathing during exercise, or worst of all open-mouth breathing as normal while inactive.

 

It was not something I had thought about but subsequently realised that I was a somewhat good nose-breather during moderate exercise.

 

From this point on I insisted upon it with myself and after the period of adaptation to it, receiving clear benefits, advised everybody who came through my door to convert to it also.

 

It varies between 3 weeks to 3 months for people to adapt to strict adherence. Douillard’s thesis is that your physiology is changed.  I thought that lactate testing should show up some of this adaptive process.

 

I experimented with it in self-training for Masters Rowing and did pretty good with results, avoiding the frequent over-training of younger years, generating the not too uncommon thoughts in one’s accumulated years of “what if I knew then what I know now?”  C’est la vie!

 

From long memory looking back, self-experimentation with this gave equally intriguing lactate results.

 

1.  In the beginning of trying this, the upper-end of maintaining nose-breathing was a very modest pace, along with expected Heart Rate.  (Many clients had to cut back to almost a fast-walk and could barely handle staying with it. A lot of them arguing against it!) Lactate at “nose-threshold” in myself then was only about 2.5 to 3 mmol.  Above this intensity I would burst into old-habit mouth-breathing.

 

2.  As the weeks went by HR at pace dropped and lactate went to about 3.5 mmol at max. nose-breathing with an increased pace at this.  The HR drop at pace was not due to any increase or other factors of altered training over that year or any other years.  That is, I had not really had a drop in HR at set paces for similar amount and level of conditioning in decades!

 

3.  After many years from 1994 “living” this way, the last surprise result was that I could maintain nose-breathing (with some will - effort) at a pace-HR that equated to 4 mmol Threshold. That was some years ago now and I remember thinking that I felt I could probably taken it up to maintain nose-breathing at just above Threshold, say 5 mmol.  Have not to this day verified if that is possible.

 

So, there is an experimental idea for a post-grad thesis!

 

There is a lot more to this but can’t put a book here!

______________________

 

All the best to you for Xmas

 

KJ & ER Goodhew.

 

 

 

 

 

Wednesday, 4th November 2015.

 

 

A relatively small amount of what is initially high intensity tolerance work repeated daily with a largely untrained individual has a dramatic adaptive effect on improved tolerance to that work as measured by lactate response.

Referring to the left hand graph, there is as said for the “tidbit” below (last week), so much in this.  Edwards (1939) is another giant in early Exercise Physiology that the profession is built on today.

 

“Fast forward”: Being the original importer-distributor of small hand-held lactate testing meters in 1995, the “Accusport” by Boehringer-Manheim, we wanted to gather as much broad information as possible on the possible varieties of use and benefits of such easy technology. In those days we offered the “BM&S Clinic” for personal training in any sport code, individual or team, and a specialised weight-loss service.  Memory from those days is that successes with people in weight-loss were at least if not more gratifying than successes with athletes!

 

One client’s success that stands above all was a 136kg woman who had “tried everything” to lose weight and despaired.  Included with “everything” were gyms and personal trainers:  Their approach was to “thrash” and basically hurt her with relentless high intensity sessions that were unpleasant.  An objective “professional” approach in weight-loss exercise prescription simply was not there in those days. (Being away from involvement in such services for about 12 years I have not really looked closely at the state of the efficacy of the “weight-loss game” today!)

 

What did we do then that was different? We lactate tested everybody who came through our door whether their goal was prospective Olympian, beginner runner wanting to do first marathon, weight-loss, triple by-pass cardiac rehab, wanting to look good for her wedding, a horse, …and even the postman! True!

 

When my favourite client story first came through the door she was scared of exercise. I told her that she would lose weight with walking at correct intensity and that it would be easy compared to what she had done and that we would work it out by testing her blood to make sure the exercise is not at unpleasant and unnecessary high intensity which puts damaging acid into your blood and through your body. That is, putting things into simple terms without off-putting sport science jargon.

 

As a prelim., with HR monitor on we set off up the road which leads to Frasers Gully about 2.4 km in length with a more challenging slope in its last ¼, but which is in the most part a barely perceivable gradual slope to find that she could not walk around 100m without bursting into rapid mouth breathing and appearing bothered.  Next time I conducted a necessarily “off-the-cuff” modified lactate assessment protocol just to find where her 4 to 5 mmol HR intensity was.

 

Several times per week we met to do Frasers Gully interval style whereby as soon as her HR climbed above her original 5mmol HR we would stop, let it drop for a minute or two and then on until the alarm called for the next stop.  

 

At the end of just one month my 136kg client who had lost about 6kg walked to the top of the gully without one stop or alarm of high HR.  Her blood pressure, borderline before, was now normal.

 

She told me she was “…amazed how easy and pleasant the exercise intensity was…” for her to achieve so much.

 

The graph on the left in today’s copy & paste reminded me of the mechanism of this favourite client success.

 

 

 

 

Friday, 23rd October 2015. :  FYI:  If you thought I was talking crazier than usual, some kind of load-up error occurred where the wrong table appeared! Corrected 25/1/2017

 

 

Classic tabulated data on responses to various brief training impulse/recovery cycles from the now classic eponymous text by Astrand and Rodahl, “Textbook of Work Physiology”.

 

Yes! I still have my copy from undergrad days!  In it is this referenced original study by Christensen et al, 1960!

 

 

 

 

 

 

 

mMol

16.7

 

 

 

 

 

 

mMol

2.6

1.8

4.9

2.2

5.7

2.3

1.8

 

 

 

I’ve added to the right an approx. conversion of mg/100ml to mMol.

 

There is so much that can be taken from this table that I cannot really get into too much here since having looked at it and making use of it in training individuals years ago.  In past days “in da game” I got a reputation for taking what people perceived as quite ordinary ranked athletes to a level unexpected.  One day watching an athlete come in a spectator said to me, “…how is that possible”? I was young then, saying somewhat flippantly, “training secrets”. This was later not taken well being accused of offering unfair advantage to the athlete over others in the club who were expected to dominate in a predicted procession of race positions. Envy amongst coaches in NZ sport is a not uncommon terrible thing; I did not last long in that club!

 

Anyway!  How did I use this table? The issue for the average athlete is that they have average aerobic capacity, average VO2max, and come up against gifted athletes with lungs and heart of a horse. What is one to do?  We know VO2 is trainable to a significant degree, but more so is % VO2 that AT kicks off a rapid lactate accumulation as determinant of endurance performance.  All we can do is optimize the average athlete’s VO2 and then shift their “threshold” as close as possible toward their VO2max.  (…plus effective race strategy, work on motivation, psychology, and structural soundness – anatomy – for freedom from potential to injury. The latter most important since however you do it, VO2max pace work is high stress work. I don’t advocate you do any of this with a beginner!) 

 

We also know that anaerobic-lactate work destroys aerobic enzymes for upto several days and opens one up to illness (immune compromise).  You can see from the table that doing VO2max pace continuously for 4 minutes takes lactate to 16.7 mMol and therefore aerobic enzymes destroyed. This is not suitable training!  Coaches who over max test or over-race their athletes in this form are simply stuffing them up!

 

You can see for an already fairly well trained athlete work/rest intervals of 10:5 and 15:10 elicit too high lactate response, destroying aerobic capacity for subsequent days. 

 

You can see the best strategy to elicit a near maximum VO2 of 5.3 with a lactate response equivalent to “going for a jog” is 15:15.

 

Experiment with this and during it wack in a lactate test or two to determine your athlete’s individual response to this regimen.

 

I can assure you this works a treat. I self-tested (when I was young with “OCD” for discerning “secrets”) and used it on numerous athletes to raise them up quickly.  If you schedule it weekly use their mood to decide whether to stick with it religiously as per schedule or not.

 

 

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Archive:

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Monday, 23rd January 2017

 

 

 

News!

 

The trend since Rio carries on!

 

We ordered 50% more Meters than we ever have with 2/3 gone either before they got here or within 2 weeks.  Amazing!  At current trend we are going to run out before next shipment gets in, don’t procrastinate!

 

Something new:

 

Class Set Wholesale:

A confidential deal on sets for educational exercise physiology (or other) labs is available:  Minimum quantity = 5 Meters.

 

Such a minimum quantity was chosen since it could meet the small number of students in labs of size 10 to 15 students with one each of tester, recorder, and subject.

 

Even Secondary “Sport Science” students could do this with tester, recorder, and roping in a subject from say the school rowing squad!

 

Just a  personal view: All those in their last year of school rowing wanting to maintain their interest and progress in the sport would have their decision making greatly helped with a clear lactate profile revealing performance improvement progress and potential trajectory which is much better shown with a lactate profile rather than all-out Erg tests which can wildly disguise capacities and potential for improvement!

 

Contact us for the Educational Quantity Deal on 5 (or more!):

 

Enquire now: Email (click here!)

 

 

 

 

Friday, 9th December 2016

 

 

News!

 

Since the Rio Olympics NZ Sport has gone to another level!

 

A notable phenomenon has occurred since the Rio Olympics:

 

We sell most of the small portable lactate testing strips in NZ, for a long time now, being since the technology was first launched and we picked it up that same year in 1995: This has afforded us a unique position of insight into who is doing what in terms of science based training in Aotearoa, particularly in endurance sports.

 

The first thing that we noticed by the millennium was that the sport that does the most lactate testing is our most successful Olympic sport, hands-down, and is still in that same position doing 21 years of regular systematic lactate assessments in NZ! An immense knowledge base is built up with all that time. It has had us asking what the other NZ endurance sports are doing for sport science. The results seem very telling to us. But, to be direct in asking about that is not a comfortable role or position for us as Supplier.

 

HOWEVER, since Rio there has occurred a phenomenon that may signal a dramatic change to this status quo:

 

Unlike each Olympics since Atlanta, USA, 1996, after these events we would endure a great drop off in demand of strips and Meters …  

 

…conclusion: Little to no lactate testing would be done post Olympics

 

…This year after Rio, the expected high demand of pre-Olympics has been superseded by higher demand AFTER the Olympics!!!

 

We’ve never had anything like it in 21 years and we’re going to have a job getting our next shipment here before we run out, since projections are to have us run out 5 months ahead of schedule!!!  We’ve got demand wrong by a month over the years but never by 5 months!

 

We’re wondering if there has been a spark of belief occur at Rio that is going to take NZ to a new level at the next Olympics?

 

Have “we” thrown off our seeming forever cringing excuse of “small nation”, with often taking the choker role at the big-time, with FINALLY NOW HAVING AN INFECTIOUS BELIEF IN OURSELVES that any athlete lining up beside us from anywhere in the world is just another athlete, and that our young talent are just going to go for it like never before?

 

…that we really are great at this thing called the Olympics?

 

I don’t know!  Anyone else have any ideas why Trainers / Sp. Scientists are right now going for it like never before? Keen to hear.

 

Whatever, it deems great for an exciting future.

 

 

 

 

 

Monday, 4th April 2016

 

We are still looking for a few more “On-sellers” (retailers) in various areas around NZ: Contact us:

Email (click here!)

 

 

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Testimonials

 

 

 

Wednesday, 19th February 2014

Shorty Clark…

…is all concentration on his way to winning Taranaki Triathlon several years ago…

…uses LP2 to good effect.

Approximately 12 months ago I started using my Lactate Pro 2 Meter for Training and Racing purposes. Previous to this I relied heavily upon Heart-rate Monitors and “subjective feel” data to ascertain my performances and success/failures.

 

The most crucial benefit I find is that it allows me to train and race way more precisely and accurately. It gives me the added advantage of knowing how to increase my Lactate Tolerances when in heavy/extended sessions or even when doing a recovery type mode work-out. I can therefore correlate my Heart-rate a lot closer to my Lactate Thresholds, thus allowing me to race smarter, quicker, efficiently and faster.

 

When racing, it’s critical not to start at to higher Heart-rate and Lactate level, as this generates into fatigue, poor performance and the inevitable bonking. By measuring and testing your Lactate levels in training, you are then able to maximise your performance and results, come race day. It’s such a valuable and necessary tool in my training and racing programme, that without it, I would be always wondering and 2nd guessing at why my performances were not at their highest level. One thing that is for sure – Blood Lactate levels DO NOT lie!!, even though you may think and feel differently.

 

The Lactate Pro 2 gives you that complete and 100% answer in how to improve your training speeds, conditions, results and performances, via the easy and simple testing methods, plus the added benefits of being simple to test, quick on results, with accuracy and lightweight to use/carry.

 

I thoroughly recommend the Lactate Pro2 Meter as the best, overall piece of training equipment that has lifted my performances, and allowed me to compete at my top potential. As a serious Age Group Triathlete, the Lactate Pro 2 has given me an added advantage to optimise my full athletic potential, and permits me to train / race in the best possible shape and condition.

 

Shorty CLARK.

Mens 60 - 64 Age Group, Tri New Zealand.

 

 

 

 

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Compare the *New* with the Old

 

 

LactatePro (1)

LactatePro2

Measurement range

0.823.3mmol/L

0.525.0mmol/L

Measuring Time

60 Sec.

15 Sec.

Sample volume

5.0μL

0.3μL

Valid period of the test strips

12 months

18 months

Calibration

using a calibration strip

Automatic calibration (No coding)

Operating Environment

1040

540

Memory

20 test results

330 test results

External output

No

Yes

Button

No

Yes

User Identification

No

Identify 3 users

 

12/11/2012

 

In Summary:  The new LP2 is 75% faster at 15 seconds, with a lower and higher lactate value measurement range, and increased temperature use range down to 5C. Its calibration is automatic, no longer requiring a calibration strip. At 18 months from date of manufacture the test strips have a 50% longer life before expiry date. The LP2 can take 330 test results in memory which will be downloadable for analysis on PC software by cable. Software & cable availability at this date yet to be announced.  The Meter can host upto 3 user identities: This could work out quite economical for a small group owning a Meter between them.

 

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Frequently Asked Questions & Answers! 12/11/2012

The LP(1) Meter production has stopped, will they stop producing the LP(1) test strips and when? We don’t know the answer to this!  Best you upgrade early!

It stands to reason that the manufacturer is giving you (…all LP users) a fair transition time to upgrade by purchasing a new LP2 Meter FROM JUNE this year!  You’ve already had 6 months to consider upgrading. If you leave it until a halt to production notice is given, and join those who panic to get one due to not being organised, then you could find we may not be able to supply you immediately with a Meter for some time when you urgently want it, joining those ranks that would cause a scarcity of supply through sudden world-wide demand!

 

This “segways” (segues) to:

Understand that when we place our orders with the factory we must predict 4 to 6 months ahead as to what we think the demand will be for any given month.  This is especially so with consumables. Most people want maximum duration to expiry, therefore we do not order in by excessive quantities for the chance to cover some exceptional (unpredictable) high demand scenarios as customers do not want stock that has been shelved too long. We can of course predict high demand for Olympic Year!  If we are hit with sudden demand above projections or the manufacturer is hit by huge demand world-wide then it is possible that we are unable to supply you for a month or more.  Therefore, You should always plan to order a minimum one month ahead of important testing schedules to avoid the 0.1% chance we could fail to supply overnight for your order.  And, you should also employ some basic management yourself by setting a minimum quantity re-order trigger strategy for test strips so that you do not completely run out!

 

Can I use the new LP2 strips in my old Meter or LP(1) strips in new LP2 Meter?  No!

Can you take the steering-wheel out of your wonderful old 1956 Volkswagen Beetle and put it in your brand new 2012 Series-5 BMW?  See “Compare” above: The LP(1) test strip measuring time is 60 sec’s, the LP2 15 sec’s; How would a 15 sec chemical reagent measuring-time strip work in a 60 sec Meter?  The LP2 sample is just 0.3 ul, the LP(1) needs almost twice as much sample volume; If you could get it in the machine the LP1 Meter would read that half volume as an “error”!  Calibration:  The LP2 requires no calibration-strip checking, this is now automatic. If you tried to put a non-automatic calibrating LP(1) strip into an LP2 Meter how would the Meter read it?  And…    you got it already!

 

Is the Software and cable included? No!

The new LP2 Meter and test strips are at exactly the same cost price-point from the manufacturer as the original LP(1) Meter and strips.  We think this is an excellent deal for you in an upgraded product that incorporates multiple superior features.  The LP(1) did not have software and connecting cable, which is a significant material addition to the original product, and is therefore of course an item which is an add-on sale to your LP2 purchase.

 

I wish to purchase the LP2 Software & Connector.  We will notify here when these are available. We do not have a date yet that these will be shipped to us.

 

What reliability and validation research do you have? …is it more accurate than the LP(1)?  We have no product comparative data yet.

The product is brand new!  We may have been the first country outside of Japan to stock and supply it.

Apart from Australia I do not know any other country with it, it is that new. We get purchase requests from Germany, South Africa, USA …!

 

I am only speculating that the product was developed and validated entirely in-house, perhaps in co-operation with Universities of Japan.  Would it not stand to reason that a new upgrade model would be more reliable, accurate, and fully validated over the original before a factory would be tooled up with tens of million$ expense and before doing production runs of it?  Take my word; from my experience with this Japanese company since 1997, you can be confident in Arkray LactatePro2 as being a superior product.

 

It is wide-open for you to make a name for yourself by doing some product comparison studies right now and being first in the world to publish on it!  Come on “kiwi” get on with it!

 

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Thursday, 19th July 2012

http://www.arkray.co.jp/english/ex/img/20120416_01.jpg

LP2

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Here (NOW!) Be the first to get one.

We already have advance orders:

Order yours NOW! 1/6/2012

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Arkray Website News Release

 

2012/04/16

 

The smallest meter in its class just got easier to use. Even better performance for ARKRAY’s card-sized blood lactate measurement device

ARKRAY, Inc. (Headquarters: Kyoto City, Nakagyo-ward), a manufacturer of sample test devices and in vitro diagnostic reagents is set to launch its new and simple blood lactate meter, the Lactate Pro™ 2 LT-1730 on 17 April. In its card size form, it is small enough to allow measurement anywhere and boasts increased performance for the rapid measurement of lactate.

 

Lactate in blood is an essential marker in the fields of sports physiology, sports science and training. The simple and rapid testing of lactate is a big benefit to both research and medicine.

To answer the needs of the market, ARKRAY released the Lactate Pro TM back in February 1997, which marked a significant reduction in size for blood lactate at the time. This new card sized device enabled simple measurement of lactate anywhere and helped to support clinical testing across many different settings.

ARKRAY is now set to release the simple blood lactate meter, the Lactate Pro™ 2 LT-1730 together with its compatible reagent/ measurement electrode, the Lactate Pro™ 2 Sensor. The measurement time has been cut by 75%* to just 15 seconds; the minimum sample volume reduced by over 90%* to just 0.3µL and the basic performance of the device improved significantly. The hassle associated with correction of reagent lots has been eliminated which also helps to reduce the risk from correction errors.

ARKRAY will continue to answer diverse testing needs in the market.

*In comparison to existing ARKRAY products:

 

http://www.arkray.co.jp/english/ex/img/20120416_01.jpghttp://www.arkray.co.jp/english/ex/img/20120416_02.jpg

Lactate Pro™ 2 LT-1730                      Lactate Pro™ 2 Sensor

 

Main features of the Lactate Pro™ 2

●A simple blood lactate device, the smallest of its kind
Palm-sized for measurement in any environment: now you can measure lactate rapidly anywhere

●Minimum sample size: just 0.3µL
Development of a new reagent sensor: measurement possible with just 1/10th the volume previously needed

●Measurement time: 15 secs
On site measurement is possible using finger-stick blood: results are shown in just 15 seconds from application of blood to the sensor- one quarter the time needed for measurement with previous devices.

●No correction needed
No more hassle from correction of sensor lots (insertion of sensor chips before measurement): this also helps to reduce the risk from correction errors.

About lactate
Blood lactate is often used in the scientific training for endurance sports such as soccer, swimming and athletics. As the intensity of the exercise increases, so too does the concentration of lactate in the blood. By measuring this change, training can be configured to delay sharp rises in blood lactate.  (There are also Medical and Equine/Veterinary applications – KJG, BM&S Imports-NZ.)

 

Product features:

 

1. Name

Lactate Analyzer Lactate Pro™ 2 LT-1730

2. Release date

17 April 2012 (Tues.)

3. Specifications:

 

Sample type

Whole blood

Meas. items

Lactate in blood

Meas. principle

LOD enzyme electrode method

Meas. range

0.5-25.0mmol/L (5-225mg/dL)

Processing speed

15 sec/ sample

Min. sample vol.

0.3uL

Compatible reagents

Lactate Pro™ 2 Sensor (Electrode for measuring lactate in blood)

Data memory

330 measurements

Ext. output function

Yes

Correction method

No correction

Temp. correction

Auto-correction using internal temperature sensor

Meas. conditions

Temp: 5-40 C, Humidity 20-80% RH (No condensation)

Power source

3V lithium battery/ CR2032 X1

Outer dimensions

50mm(W) X 12mm(D) X 100mm(H)

Weight

Approx. 45g (Incl. batteries)

 

Product method

Description: Description: Description: Description: Description: Description: http://www.arkray.co.jp/english/ex/img/20120416_03.jpg

 

 

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We are an independent importer-distributor.  No middle-man!  ...we import directly from the manufacturer.  Our prices offered to you are always at the lowest possible mark-up being wholesale direct rates, often equal to or BELOW the lowest price found anywhere in the world (ensuring correct exchange rate, shipping and GST have been accounted for!).  Even though our mark-up is small we do offer to split and share it with a number of businesses who are enthusiastic about lactate testing, who on-sell (retail) to their customers.

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~ Product List ~

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Lactate Pro

Description: Description: Description: Description: Description: Description: LT-1710(H)_1

Most popular in NZ

 

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Support Information

Index:

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Medical

-       Obstetrics-Maternity

 

-       A&E

 

1.                 This article posted here 26 Nov. 08 cited from HIV InSite, University of California, San Francisco.

 

Point-of-care lactate testing predicts mortality of severe sepsis in a predominantly HIV type 1-infected patient population in Uganda

UCSF Institute for Global Health Literature Digest
Published April 17, 2008

Journal Article
Moore CC, Jacob ST, Pinkerton R, Meya DB, Mayanja-Kizza, Reynolds SJ, et al.

Point-of-care lactate testing predicts mortality of severe sepsis in a predominantly HIV type 1-infected patient population in Uganda. Clin Infect Dis 2008 Jan 15; 46(2):215-22.

Objective
To evaluate the ability of a handheld portable whole-blood lactate (PWBL) analyzer to predict mortality in patients who are admitted to the hospital with severe sepsis.
Study Design
A prospective observational study.
Setting
An accident and emergency department of Mulago Hospital, a national referral hospital in Kampala, Uganda.
Participants
72 patients were enrolled in the study. Inclusion criteria were ≥18 years of age and admission to a medical ward, along with: 1. two or more systemic inflammatory response syndrome criteria (body temperature, >38°C or <36°C; heart rate, >90 beats/min; respiratory rate, >20 breaths/min; or peripheral WBC concentration, >12,000 cells/mm3 or <4000 cells/mm3; or thermodysregulation; 2. systolic blood pressure ≤100 mm Hg; and 3. a suspected infection. Exclusion criteria included acute cerebrovascular events, gastrointestinal hemorrhage, or admission to the surgical or obstetrics and gynecology ward.
Intervention
There was no intervention in this study. This analysis was conducted among a subset of 253 patients recruited to study the incidence, management, and outcomes of sepsis. From this sample, the first and last 50 consecutively enrolled patients were recruited to determine the predictive value of PWBL in predicting mortality from sepsis. Background information - including age, sex, HIV-1 serostatus, and prescribed antiretroviral medicines (ARVs)-was recorded. At patient enrollment, temperature, heart rate, respiratory rate, and blood pressure were measured. To determine outpatient survival, an attempt was made to telephone patients 30 days after their discharge from the hospital. A rapid HIV-1 test and malaria smear were performed at Mulago Hospital. A local private clinical laboratory provided results of lactate and bicarbonate analysis. PWBL was obtained using a lancet to collect a drop of whole blood from the patient's finger, for analysis by a handheld portable device. This instrument uses enzymatic determination and reflectance photometry of lactate in the plasma portion of whole blood using a measurement strip. Standard laboratory serum lactate (SLSL) concentration was obtained by phlebotomy for venous blood samples. Within 2 hours of sample collection, the sample was transported in a standard serum tube via a cooler to the clinical laboratory, where blood was centrifuged and serum was removed for use in the lactate assay.
Primary Outcomes
In-hospital mortality.
Results
Information was available for 72 of the 100 enrolled subjects. The mean age of participants was 35.7, 61.1% were women, and 81.9% were HIV infected, with a mean CD4 lymphocyte count of 88.6 cells/ mm3. These 72 patients were similar to the larger study population in age (mean age, 35.7 vs. 33.8 years), sex (61.1% vs. 59.1% female), HIV-1 seropositivity (81.9% vs. 86.6%), and ARV status (13.9% vs. 10.6% ARVs prescribed). Fifty-nine (81.9%) of 72 evaluated patients were infected with HIV-1. The in-hospital mortality rate was 25.7% (18 of 70), and the in- and outpatient mortality at 30 days was 41.6% (30 of 72). PWBL was positively associated with in-hospital but not outpatient mortality (p <.001). The receiver operating characteristic (ROC) area under the curve for PWBL was 0.81 (p <.001). The optimal PWBL concentration for predicting in-hospital mortality (sensitivity, 88.3%; specificity, 71.2%) was ≥4.0 mmol/L. Patients with a PWBL concentration ≥4.0 mmol/L died while in the hospital substantially more often (50.0%) than did those with a PWBL concentration <4.0 mmol/L (7.5%) (odds ratio, 12.3; 95% confidence interval, 3.5-48.9; p <0.001). SLSL levels were lower among survivors than among deceased. The ROC under the curve for predicting in-hospital mortality was 0.72 (p=0.004). SLSL results were inconsistent and less predictive of mortality than were those of PWBL.
Conclusions
The authors conclude that PWBL testing can quickly identify patients who require immediate interventions,
and it should be included in evaluation and treatment algorithms for septic patients. PWBL testing could be used in village health posts, for earlier transfer of septic patients to facilities with a higher level of care, and in referral hospitals, for triage of patients to acute care settings where appropriate resuscitation can begin.
In Context
Elevated lactate concentrations and poor clearance of lactic acid are known to increase mortality from severe sepsis. Lactate concentrations, combined with other laboratory measures, are used to guide early management of sepsis. Measurement of lactate concentration in developing countries is difficult because of limited resources. An effective, inexpensive method to measure lactate concentration without sophisticated laboratory resources has the potential to offer a method to identify patients in greatest need of rapid, aggressive treatment of sepsis.
Programmatic Implications
The use of the PWBL and its value in predicting mortality from sepsis in populations with high prevalence of HIV has potential to assist health care workers in resource limited settings in prioritizing these patients. Hospitalization, availability of support for potential multisystem failure, and rapid administration of antimicrobial agents can be delivered more urgently in persons with high lactate concentrations.

 

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2.                 This article posted here 26 Nov. 08 cited from Lab Tests On-line.

Lactate Testing in Acute Assessement

What is being tested?
This test measures the amount of lactate in the blood or, more rarely, in the cerebrospinal fluid. Lactate is the ionic (electrically charged) form of lactic acid. It is produced by muscle cells, red blood cells, brain, and other tissues during anaerobic energy production and is usually present in low levels in the blood. Aerobic energy production is the body’s preferred process, but it requires an adequate supply of oxygen. Aerobic energy production occurs in the mitochondria, tiny power stations inside each cell of the body that use glucose and oxygen to produce ATP (adenosine triphosphate), the body’s primary source of energy.

When cellular oxygen levels are decreased, however, and/or the mitochondria are not functioning properly, the body must turn to less efficient anaerobic energy production to metabolize glucose and produce ATP. In this process, the primary byproduct is lactic acid, which can build up faster than the liver can break it down. When lactic acid levels increase significantly in the blood, the affected person is said to have first hyperlactatemia and then lactic acidosis (LA). The body can often compensate for the effects of hyperlactatemia, but LA can be severe enough to disrupt a person’s acid/base (pH) balance and cause symptoms such as muscular weakness, rapid breathing, nausea, vomiting, sweating, and even coma.

Lactic acidosis is separated into two types: A and B. Type A may be due to inadequate oxygen uptake in the lungs and/or to decreased blood flow (hypoperfusion) resulting in decreased transport of oxygen to the tissues. The most common reason for this is shock from a variety of causes including trauma and blood loss, but LA may also be due to conditions such as heart attack, congestive heart failure, and pulmonary edema (fluid in the lungs). Type B is caused by conditions that increase the amount of lactate/lactic acid in the blood but are not related to a decreased availability of oxygen. This includes liver and kidney disease, diabetes, leukemia, AIDS, glycogen storage diseases (such as glucose-6-phosphatase deficiency), drugs and toxins, severe infections (both systemic sepsis and meningitis), and a variety of inherited metabolic and mitochondrial diseases (forms of muscular dystrophy that affect normal ATP production). Strenuous exercise can also result in increased blood levels of lactate.

 

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3.                 Green Prescription

 

1.  Lactate Testing in General Practise

With the advent of the world’s first small hand-held lactate test meter launched in 1994 by Boehringer Mannheim, the use of lactate prescription for effective and objective “Green Prescription” was already well considered.  My personal observation is that the Europeans are well ahead of us in this medically correct and safety orientated approach to intensity prescription of exercise.  It is this correct medical approach that has been begging for implementation here in NZ. Fourteen years later (2008) the right Doctor with knowledge & motivation to champion this cause here is yet to be forthcoming!

 

To view larger image in viewer on your PC click here.

Description: Description: Description: Description: Description: Description: Lactate in GP practise.jpg

 

2. This article posted here 26 Nov. 08 cited on-line at Wiley Interscience.

 

Interval versus continuous training in patients with severe COPD: a randomized clinical trial  Eur Respir J 1999; 14: 258–263.

R. Coppoolse*, A.M.W.j. Schols*, E.m. Baarends*, R. Mostert**, M.a. Akkermans**, P.p. Janssen**, E.F.m. Wouters*

**Astmacentre Hornerheide, and   *Dept of Pulmonology, Maastricht University, the Netherlands.

Correspondence: A.M.W.J. Schols
Dept of Pulmonology
P.O. Box 5800
6202 AZ Maastricht
The Netherlands
Fax: 31 433875051

Copyright Copyright ERS Journals Ltd 1999

Interval versus continuous training in patients with severe COPD: a randomized clinical trial. R. Coppoolse, A.M.W.J. Schols, E.M. Baarends, R. Mostert, M.A. Akkermans, P.P. Janssen, E.F.M. Wouters. ©ERS Journals Ltd 1999.

ABSTRACT

 

Abstract

Limited information is available regarding the physiological responses to different types of exercise training in patients with severe chronic obstructive pulmonary disease (COPD). The aim of this study was two fold: firstly, to investigate the physiological response to training at 60% of achieved peak load in patients with severe COPD; and secondly to study the effects of interval (I) versus continuous (C) training in these patients.

Twenty-one patients with COPD (mean±sd forced expiratory volume in one second: 37±15% of predicted, normoxaemic at rest) were evaluated at baseline and after 8 weeks' training. Patients were randomly allocated to either I or C training. The training was performed on a cycle ergometer, 5 days a week, 30 min daily. The total work load was the same for both training programmes.

C training resulted in a significant increase in oxygen consumption (V 'O2) (17%, p<0.05) and a decrease in minute ventilation (V 'E)/V 'O2 (p<0.01) and V 'E/carbon dioxide production (V 'CO2) (p<0.05) at peak exercise capacity, while no changes in these measures were observed after interval training. During submaximal exercise a significant decrease was observed in lactic acid production, being most pronounced in the C-trained group (-31%, p<0.01 versus -20%, p<0.05). Only in the I-trained group did a significant increase in peak work load (17%, p<0.05) and a decrease in leg pain (p<0.05) occur. Training did not result in a significant improvement in lung function, but maximal inspiratory mouth pressure increased in both groups by 10% (C: p<0.05) and 23% (I: p<0.01).

The present study shows a different physiological response pattern to interval or continuous training in chronic obstruction pulmonary disease, which might be a reflection of specific training effects in either oxidative or glycolytic muscle metabolic pathways. Further work is required to determine the role of the different exercise programmes and the particular category of patients for whom this might be beneficial.

 

 

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Equine Trainer & Veterinary

 

Books

Allan Davie is probably the pre-eminent advocate in Australasia of effective training of race horses incorporating lactate testing.  The great value of his book is the simplicity of its explanation.  Anybody can use it to catapult the training of their horses into 21st Century method to earn positive performance improvements.

 

Description: Description: Description: Description: Description: Description: DavieCover.gif

 

AU$35

Price NZ$ relative to currency x-rate!

Articles

Bad news for heavy riders and narrow horses

March 3, 2008

Researchers in the US have bad news for overweight horse riders. A study has found that horses that have to carry between 25 and 30 per cent of their bodyweight have more physical problems related to exercise than those who carry 20 percent or less.

Horses carrying 30% body weight showed a significant increase in muscle soreness and muscle tightness scores. The changes were less marked when they carried 25% body weight.

Dr Debra Powell .... conducted a study ....performing a standardised ridden exercise test in an indoor school arena. After five minutes active walk to warm up, the horses were ridden at a trot (3m/s) for 4.8km, followed by 1.6km at a canter (5m/s). This exercise schedule was chosen to simulate a 45-minute work period of work typical of an intermediate-level riding school horse.

The researchers measured heart rate, plasma lactate concentration and creatine kinase. Lactate is produced in the muscles during exercise. At low levels of work the body can metabolise it and so levels in the plasma remain low. As the work level increases the rate of lactate production exceeds the body's ability to remove it and so concentrations rise. Creatine kinase (CK), an enzyme present in the muscles, is released into the blood as a result of some types of muscle damage.

... Plasma lactate levels were higher immediately after exercise and 10 minutes after end of exercise, in horses that carried 30% of their body weight....

...(more)...  http://www.horsetalk.co.nz/news/2008/03/011.shtml

 

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Health, Fitness & Sport

 

Books:

 

 

 

 

 

 

 

 

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FIND A TEST SERVICE PROVIDOR IN YOUR LOCAL AREA.

 

NOTICE TO SERVICE PROVIDORS:  Opt in or out list!  Apologies if you are surprised to be not listed! This listing service has only been launched since Monday 3rd November 2008.  It is an opt-on, opt-off list.  That is, let me know if you want to be on it.  If your business or interests have changed please let me know when you do not want referrals to continue.

 

 

Whangarei

NorthTec - Northland Polytechnic

Ady Ngawati

Ph 459 5241

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Ph 459 5251

BM&S Product Services: Lactate

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Joe McQuillan

Millenium Institute of Sport & Health; TriathlonNZ Support Services

Ph 524 6957

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Waikato Institute of Technology

Adrian Pooley

Ph 834 8800 x 8658; 021 1157 7457

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Palmerston North

IFNHH Faculty, Massey University

Matt Barnes

Human Performance Lab

Ph 356 9099 x 7637

http://ifnhh.massey.ac.nz/sportexercise/

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Wellington

 

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Andrew Jamieson

Ph 0800 21 FitLab (21 348 522) ; 021 348 847

www.fitlab.co.nz

BM&S Product Services: Lactate; TANITA InnerScan Segmental Body Composition Analysis.

 

Christchurch

College of Education, University of Canterbury

Gavin Blackwell

Ph 345 8173

BM&S Product Services: Lactate; Cosmed K4b2 – VO2 max etc.

 

Canterbury University – Recreation Services

Stephen Rickerby

Ph 03 364 2987 x 8650

BM&S Product Services: Lactate

 

Queenstown

Sportbase Ltd

Sam Thompson

Ph 021 921 114

www.sportbase.co.nz

BM&S Product Services: Lactate Testing

Preferred Client:

 

Frankton

Proactive Physiotherapy Ltd

Sonya Anderson

Ph 03 442 7667

BM&S Product Services: Lactate Testing; TANITA InnerScan Segmental Body Composition Analysis.

Preferred Client:

 

 

Dunedin

Exponential Performance Coaching

Matty Graham

Ph 027 384 1127

http://exponentialperformance.blogspot.com/

BM&S Product Services: Lactate Testing;

 

 

Invercargill

SIT – School of Health Exercise & Recreation

Damian Tippen

Ph 211 2699 x 8744

BM&S Product Services: Lactate

 

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www.kitchenscales.net.nz

take a look here at NZ’s top-selling Kitchen scale

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